PK Protocol
The PK Protocol aids in regeneration of the cell membrane to treat neurotoxicity in order to treat the following:
-ALS, Motor Neuron Disease
-Alzheimer’s
-Anti-Aging, Longevity
-Autism and Developmental Delay
-Cancer
-Cardiovascular Disease
-Chronic Fatigue Syndrome
-Environmental Illness
-Epilepsy
-Heavy metal detoxification
-Hepatitis C
-Huntington’s Disease
-Hyperlipidemia (elevated cholesterol, LDL, triglycerides)-Infertility
-Inherited Metabolic Disorders
-Lyme Disease
-Mold Toxicity
-Multiple Sclerosis
-NeuroLyme
-Neurotoxicity
-PANDAS
-Parkinson’s
-Post Stroke
-Pesticide/Chemical Poisoning
-Seizures
How The Protocol Is Implemented:
- The patient comes into the office for a blood draw for the following tests: blood chemistry and Red Blood Cell Analysis, which is sent to the Kennedy Kreiger Institute for an in-depth analysis.
- Your blood test results will then be sent to BodyBio for analysis that includes screening for Cardiac Risk, Cellular Distortion, Electrolyte Stability, Gastrointestinal Function, Hematology, Hepatic Function, Immune Status, Lipid Metabolism, Muscular Involvement, Nitrogen Retention, Psychological Stability, Physiological Stability, Pulmonary Function, Renal Function and Toxicity Status. Each of these systems will be reviewed to isolate the areas requiring intervention.
- You will then receive a personalized Fatty Acid Report, Fatty Acid Analysis, IV Protocol and Nutrient Protocol--recommendations for supplements such as electrolytes, minerals, enzymes, fatty acids, vitamins and amino acids. These protocols are tailored to you to address your specific needs.
- You will meet with Dr. DeLuca to review your reports and protocols and receive a personalized treatment plan.
For More Information:
The Patricia Kane (PK) Protocol and BodyBio Testing
BodyBio Inc. is one of only a few organizations in the world that has been able to keep pace with the available medical research on the implications of nutrition on functions and disorders. Even more importantly, BodyBio has developed proven methods (our diagnostic BodyBio Reports) to bridge the knowledge gap between applied nutritional research, health care practitioners and patients interested in nutrition as a component of their health care.
The PK --Membrane Stabilizing-- Protocol for Neurological Disorders
Background: The lipid soluble nature of toxicity has led us to seize the complexity of neurological presentations by addressing them from a cell membrane perspective. Examination of red cell lipids and identification of nuclear DNA adducts as chemicals, pesticides, microbials and metals in subjects with Motor Neuron Disease, Autism, Multiple Sclerosis, Post Stroke, Epilepsy, Alzheimer’s and Parkinson’s Disease over the past eleven years in 9000 analyses has revealed a characteristic accumulation of very long chain fatty acids (VLCFAs), which comprise lipid rafts, or ceramides, revealing cell membrane derangement. Membrane phospholipid abnormalities with elevation of VLCFAs may be indicative of exposure to fat soluble neurotoxins resulting in suppressed peroxisomal beta oxidation of VLCFAs. Disturbances in methylation due to toxic exposure may destabilize the membrane phospholipid structure and alter DNA expression due to deficits in the enzymes Methylene Tetrahydrofolate Reductase (MTHFR) and Methionine Synthase.
Objective: The use of oral and IV lipids may facilitate stabilization of phospholipids in cellular membranes thereby addressing cell membrane integrity. The addition of intravenous phenylbutrate addresses neuroinflammation by increasing the beta oxidation of VLCFAs.
Procedure: To clear the bioaccumulation of toxins and stabilize membrane function we have embarked on a clinical treatment plan for the past five years to address the accumulation of aberrant lipids and ceramides with oral and IV phenylbutyrate, bolus intravenous phosphatidylcholine as Essentiale, methylation factors (folinic acid, riboflavin, methylcobalamin, tetrahydrobiopterin), and sulfation support (IV Glutathione).
Results: We have noted significant and sustained clinical neurological improvement within the first few weeks after initiation of oral and intravenous treatment in our patient population of 300 subjects.
Conclusion: The administration of Phosphatidylcholine, Leucovorin/Folinic acid, Glutathione and Phenylbutyrate may offer a new therapeutic strategy for neurological disorders involving neurotoxic exposure.
For more information please visit www.BodyBio.com and www.PKProtocol.com
Objective: The use of oral and IV lipids may facilitate stabilization of phospholipids in cellular membranes thereby addressing cell membrane integrity. The addition of intravenous phenylbutrate addresses neuroinflammation by increasing the beta oxidation of VLCFAs.
Procedure: To clear the bioaccumulation of toxins and stabilize membrane function we have embarked on a clinical treatment plan for the past five years to address the accumulation of aberrant lipids and ceramides with oral and IV phenylbutyrate, bolus intravenous phosphatidylcholine as Essentiale, methylation factors (folinic acid, riboflavin, methylcobalamin, tetrahydrobiopterin), and sulfation support (IV Glutathione).
Results: We have noted significant and sustained clinical neurological improvement within the first few weeks after initiation of oral and intravenous treatment in our patient population of 300 subjects.
Conclusion: The administration of Phosphatidylcholine, Leucovorin/Folinic acid, Glutathione and Phenylbutyrate may offer a new therapeutic strategy for neurological disorders involving neurotoxic exposure.
For more information please visit www.BodyBio.com and www.PKProtocol.com
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